Diagnosis of Heart attack
Diagnostic Test list for Heart attack:
The list of medical tests
mentioned in various sources as
used in the diagnosis of Heart attack
includes:
Heart attack Diagnosis: Book Excerpts
Tests and diagnosis discussion for Heart attack:
Facts About Heart Disease and Women: NHLBI (Excerpt)
Doctors also have a new fast test for a heart attack. It (Source: excerpt from Facts About Heart Disease and Women: NHLBI)
Testing For A Heart Attack: NHLBI (Excerpt)
Once you get help, you will undergo tests to see if a heart
attack has actually occurred. Some tests are done at the hospital,
while others can be done by emergency medical personnel who come in
an ambulance. (Tests and
treatments done by emergency medical personnel.)
Key heart attack tests are:
|
Electrocardiogram (ECG or EKG). This is a
graphic record of the electrical activity of the heart as it
contracts and relaxes. The ECG can detect abnormal heartbeats,
some areas of damage, inadequate blood flow, and heart
enlargement.
|
|
Blood test. A blood test will be done
routinely to check for enzymes or other substances that are
released when cells begin to die. These are "markers" of the
amount of damage to your heart.
|
|
Nuclear scan. This test shows areas of the
heart that lack blood flow and are damaged. It also can reveal
problems with the heart’s pumping action. A small amount of
radioactive material is injected into a vein, usually in the
arm. A scanning camera positioned over the heart records
whether the nuclear material is taken up by the heart muscle
(healthy areas) or not (damaged areas). The camera also can
evaluate how well the heart muscle pumps blood. This test can
be done during both rest and exercise, enhancing the
usefulness of its results.
|
|
Coronary angiography (or arteriography).
This test is used to check blockages and narrowed areas inside
coronary arteries. A fine tube (catheter) is threaded through
an artery of an arm or leg up into the heart. A dye that shows
up on X ray is then injected into the blood vessel, and the
vessels and heart are filmed as the heart pumps. The picture
is called an angiogram or
arteriogram. |
(Source: excerpt from
Testing For A Heart Attack: NHLBI)
Diagnosis of Heart attack: medical news summaries:
The following medical news items
are relevant to diagnosis and misdiagnosis issues for Heart attack:
Diagnostic Tests for Heart attack: Online Medical Books
16 MEDICAL BOOKS ONLINE!
Review excerpts from medical books online, free, without registration,
for more information about diagnostis of Heart attack.
CHEST PAIN:
Ask the following questions:
(Algorithmic Diagnosis of Symptoms and Signs)
- Is the chest pain acute or chronic? If it is acute, one must consider acute myocardial infarction, pulmonary embolism, pneumothorax, pericarditis, and fractures. If the chest pain is chronic, one must consider chronic coronary insufficiency, esophagitis, hiatal hernia, and various chest wall conditions.
- Is the pain constant or intermittent? Constant pain suggests acute myocardial infarction, pulmonary infarction, dissecting aneurysm, and pneumonia. Intermittent pain would suggest coronary insufficiency, Tietze's disease, and DaCosta's syndrome.
- Is there associated significant hypertension? Significant hypertension would make one think of dissecting aneurysm, but it is also found occasionally in acute myocardial infarction.
- Is the pain relieved by antacids? Relief by antacids should prompt one to consider esophagitis and hiatal hernia.
- Is the pain precipitated or increased by breathing? The pain of pleurisy, costochondritis, rib fractures, and pneumothorax is precipitated or increased by breathing.
- Is there associated hemoptysis? Hemoptysis should make one consider a pulmonary embolism.
- Is there fever and purulent sputum? Fever and purulent sputum should make one consider pneumonia.
- Is there dyspnea? Dyspnea should make one consider pneumothorax, pulmonary embolism, and pneumonia, as well as congestive heart failure secondary to acute myocardial infarction.
- Is it aggravated by movement? Aggravation of the chest pain by movement should suggest pericarditis. Remember, myocardial infarctions may also have extension into the pericardium and must be considered at times.
- Is it relieved by nitroglycerin? Relief by nitroglycerin should suggest a coronary insufficiency, but esophagospasm may be relieved by nitroglycerin also.
DIAGNOSTIC WORKUP
All patients should have a CBC, sedimentation rate, chemistry panel, VDRL test, chest x-ray, and EKG. If there is sputum, a smear and culture should be done as soon as possible.
If a myocardial infarction is suspected, then serial EKGs and tests for the isoenzyme of creatine kinase (CK-MB) should be done if the initial EKG and enzymes do not show any significant changes. Serum cardiac troponin levels may also be diagnostic of a myocardial infarct. Thallium-201 scintigraphy is useful in diagnosing both myocardial infarction and coronary insufficiency. Exercise tolerance tests may help diagnose coronary insufficiency. Immediate coronary angiography should be undertaken if the condition deteriorates. This can be followed by immediate balloon angioplasty, reperfusion therapy, or bypass surgery.
If a pulmonary embolism is suspected, arterial blood gases and a ventilation-perfusion scan should be done.
d
-dimer testing of whole blood is a sensitive test of pulmonary embolus. Pulmonary angiography may need to be done if these are negative and pulmonary embolism is still strongly suspected.
If esophageal disease is suspected, an upper GI series with esophagogram should be done; this can be followed with esophagoscopy and gastroscopy if needed. A Bernstein test (acid perfusion of the esophagus) may reproduce the exact pain and distinguish esophageal reflux from a cardiac source of the pain. Ambulatory pH monitoring may also diagnose reflux esophagitis.
If pericarditis is suspected, echocardiography and possibly a CT scan of the chest and pericardium may be necessary. Coronary angiography may be necessary to diagnose coronary insufficiency. Echocardiography is also helpful in diagnosing mitral valve prolapse and the various myocardiopathies. Twenty-four-hr Holter monitoring is useful in diagnosing many causes of intermittent chest pain.
Referral to a cardiologist or pulmonologist may be appropriate at any point in this workup. Dissecting aneurysm may be confirmed by a CT scan or MRI of the chest.
» READ BOOK EXCERPT ONLINE »
Source: Algorithmic Diagnosis of Symptoms and Signs, 2003
Chest Pain:
Differential Diagnosis
(In a Page: Signs and Symptoms)
-
Cardiovascular etiologies
–Myocardial infarction
–Angina
–Acute coronary syndrome
–Pulmonary embolus
–Pericarditis
–Arrhythmias
–Mitral valve prolapse
–Aortic stenosis
–Aortic dissection
–Cardiac tamponade
-
Pulmonary etiologies
–Pneumonia
–COPD
–Asthma
–Pneumothorax
–Tension pneumothorax
–Hemothorax
–Empyema
–Pneumomediastinum
–Lung cancer
-
Gastrointestinal etiologies
–Esophagitis/GERD
–Gastritis
–Peptic ulcer disease
–Perforated ulcer
–Esophageal spasm
–Pancreatitis
–Esophageal rupture
–Pneumoperitoneum
-
Musculoskeletal etiologies
–Muscle strain or spasm
–Intercostal muscle spasm
–Costochondritis
–Trauma (e.g., rib fracture)
-
Zoster
-
Cancer (e.g., lymphoma)
-
Panic disorder
-
Less common etiologies include Tietze's syndrome, Pott's disease (tuberculosis of the spine), xyphodenia, cholecystitis, peritonitis, liver cancer, and hepatitis
Workup and Diagnosis
- History and physical examination
–Assess onset, duration, location, radiation, type of pain, and exacerbating and alleviating factors
–Cardiovascular evaluation includes assessment of heart sounds, murmurs, gallops or rubs, and carotid bruit
–All patients require a rectal exam (e.g., to assess for occult bleeding due to GI etiologies, to assess for occult bleeding before initiating anticoagulation)
–Risk factors for coronary artery disease include smoking, hyperlipidemia, diabetes, and a personal or family history of coronary artery disease
-
Initial evaluation may include pulse oximetry, CBC, electrolytes, BUN/creatinine, calcium, glucose, PT/INR/PTT, ECG, chest X-ray, and cardiac enzymes
-
Patients with suspected coronary artery disease may require stress testing, echocardiogram, and/or cardiac catheterization
-
Further studies to consider include arterial blood gas, liver function tests, amylase and lipase, CT of chest and abdomen, VQ scan, peak flow testing and pulmonary function tests, arteriogram, bronchoscopy, EGD, and/or esophagram
-
Transesophageal echocardiogram and/or CT scan or MRI of the chest may be required to rule out aortic dissection (if widened mediastinum is present on X-ray)
» READ BOOK EXCERPT ONLINE »
Source: In a Page: Signs and Symptoms, 2004
Chest Pain:
Differential Diagnosis
(In A Page: Pediatric Signs and Symptoms)
- Musculoskeletal
–Sharp, stabbing pain that is usually very well localized, often worsened by deep breath or cough
–Costochondritis: Tender parasternal pain at insertion of ribs into cartilage en route to sternum; increases with palpation or mild chest compression (possibly postviral)
–Injury to chest wall
- Pulmonary
–Very common cause, usually associated with respiratory symptoms: Shortness of breath, cough, exercise intolerance
–Asthma (most common), often only EIA; may have personal/family history of atopy (asthma, eczema, seasonal allergies); shortness of breath is usually primary complaint, with feeling of chest tightness/pain as a secondary symptom
–Pleuritic chest pain: Sharp, stabbing pain with deep breaths, indicates pleural space inflammation, probably postinfectious (especially viral)
–Pneumonia: Chest pain secondary to cough or pleural involvement
–Pneumothorax can occur spontaneously, especially in tall, thin athletes
- Gastrointestinal
–GERD and PUD: Burning, substernal pain with eating, worse at night
–Rarely pancreatitis (with back pain too), cholecystitis, hiatal hernia, hepatitis
- Cardiac: Rare in children
–Precordial catch syndrome: Sharp, brief (seconds) chest pain usually associated with rising from lying or sitting; unclear etiology, but of no significance
–Pericarditis: Inflammation of the pericardium; often postviral, may represent connective tissue/autoimmune, cancer, bacterial infection (very ill appearing with fever), or post-cardiac surgery; patients often lean forward to decrease the pain
–MI (rare): Congenital coronary anomaly, post-Kawasaki, cocaine use, hypertrophic cardiomyopathy
–Aortic dissection: Consider if features or history of Marfan syndrome is present
Workup and Diagnosis
- History
–Activity at onset, (chest pain with exercise is a red flag!), precipitating/relieving factors, quality of pain (sharp vs dull)
–Associated symptoms (shortness of breath, diaphoresis, cough/wheeze, nausea/vomiting), recent illness, response to eating, sleeping, different foods (caffeine, chocolate, spicy, or high-fat foods)
–Personal/family history of asthma, allergies, eczema
–Recent diagnosis of heart disease or death in a family member often generates fear in the patient or parent, prompting the evaluation of chest pain
–Social history: Recent life stressors (school problems, family discord, etc.); drug use, especially cocaine
-
Physical exam
–Reproducible with palpation likely musculoskeletal
–Chest exam: Wheezing, rales, crepitus
–Cardiac exam: Usually normal, even with cardiac
causes; pericarditis is associated with rub
-
Chest X-ray for infiltrates, pneumonia, pneumothorax
-
ECG and cardiac enzymes are rarely required but relatively inexpensive and readily available, and can rule out MI and provide reassurance for families
-
Cardiac stress test
–Continuous ECG monitoring while the patient exercises to evaluate for coronary insufficiency
–Used for patients with exercise-induced chest pain and/or coronary abnormalities
» READ BOOK EXCERPT ONLINE »
Source: In A Page: Pediatric Signs and Symptoms, 2007
CHEST PAIN:
Approach to the Diagnosis
(Differential Diagnosis in Primary Care)
A possible myocardial infarction must be the first consideration in all adults with acute chest pain especially if there are significant alterations of the vital signs. Consequently, serial ECGs, serial cardiac enzymes, and hospitalization will often be necessary. Once this condition has been excluded, we can turn our attention to the other possibilities. Arterial blood gases, chest x-ray, and a lung scan may be ordered to exclude a pulmonary embolism. Pulmonary angiography may be necessary in some cases. A chest x-ray may be ordered to rule out pneumonia. Acute chest pain related to esophagitis is often relieved by swallowing lidocaine viscus, an extremely useful tool in the differential diagnosis. Relief of the pain with nitroglycerin under the tongue or by spray will support the diagnosis of coronary insufficiency. Tenderness of the costochondral junctions with relief on lidocaine injection into the point of maximum tenderness suggests Tietze syndrome (costochondritis). In cases of chronic chest pain, an exercise tolerance test with thallium scan should be done to rule out coronary insufficiency or myocardial infarct. It may be wise to do immediate coronary angiography if the condition deteriorates so that balloon angiography, bypass surgery, or reperfusion therapy may be initiated. Dissecting aneurysm is revealed by CT scan or MRI of the chest.
» READ BOOK EXCERPT ONLINE »
Source: Differential Diagnosis in Primary Care, 2007
Chest pain:
History and physical examination
(Handbook of Signs & Symptoms (Third Edition))
If the chest pain isn't severe, proceed with the history. Ask if the patient feels diffuse pain or can point to the painful area. Sometimes a patient won't perceive the sensation he's feeling as pain, so ask whether he has any discomfort radiating to his neck, jaw, arms, or back. If he does, ask him to describe it. Is it a dull, aching, pressurelike sensation? A sharp, stabbing, knifelike pain? Does he feel it on the surface or deep inside? Find out whether it's constant or intermittent. If it's intermittent, how long does it last? Ask if movement, exertion, breathing, position changes, or eating certain foods worsens or helps relieve the pain. Does anything in particular seem to bring it on?
Review the patient's history for cardiac or pulmonary disease, chest trauma, intestinal disease, or sickle cell anemia. Find out which medications he's taking, if any, and ask about recent dosage or schedule changes.
Take the patient's vital signs, noting tachypnea, fever, tachycardia, oxygen saturation, paradoxical pulse, and hypertension or hypotension. Also, look for jugular vein distention and peripheral edema. Observe the patient's breathing pattern, and inspect his chest for asymmetrical expansion. Auscultate his lungs for pleural friction rub, crackles, rhonchi, wheezing, or diminished or absent breath sounds. Next, auscultate for murmurs, clicks, gallops, or pericardial friction rub. Palpate for lifts, heaves, thrills, gallops, tactile fremitus, and abdominal masses or tenderness. (See Chest pain: Common causes and associated findings, pages 136 and 137.)
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Signs & Symptoms (Third Edition), 2006
Pulse, absent or weak:
History and physical examination
(Handbook of Signs & Symptoms (Third Edition))
If you detect an absent or a weak pulse, quickly palpate the remaining arterial
pulses to distinguish between localized or generalized loss or weakness. Then quickly check the patient’s other vital signs, evaluate his cardiopulmonary status, and obtain a brief history. Based on your findings, proceed with emergency interventions. (See Managing an absent or a weak pulse, pages 506 and 507.)
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Signs & Symptoms (Third Edition), 2006
Coronary artery disease:
Diagnosis
(Professional Guide to Diseases (Eighth Edition))
The patient history — including the frequency and duration of angina and the presence of associated risk factors — is crucial in evaluating CAD. Additional diagnostic measures include the following:
❑ Electrocardiogram (ECG) during angina may show ischemia and, possibly, arrhythmias such as premature ventricular contractions. ECG is apt to be normal when the patient is pain-free. Arrhythmias may occur without infarction, secondary to ischemia.
❑ Treadmill or exercise stress test may provoke chest pain and ECG signs of myocardial ischemia.
❑ Coronary angiography reveals coronary artery stenosis or obstruction, possible collateral circulation, and the arteries’condition beyond the narrowing.
❑ Myocardial perfusion imaging with thallium-201, Cardiolite, or Myoview during treadmill exercise detects ischemic areas of the myocardium, visualized as “cold spots.’’
❑ Stress echocardiography may show wall motion abnormalities.
❑ Electron-beam computed tomography identifies calcium within arterial plaque; the more calcium seen, the higher the likelihood of CAD.
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Diseases (Eighth Edition), 2005
Chest pain:
History and physical examination
(Professional Guide to Signs & Symptoms (Fifth Edition))
If the chest pain isn’t severe, proceed with the history. Ask if the patient feels diffuse pain or can point to the painful area. Sometimes a patient won’t perceive the sensation he’s feeling as pain, so ask whether he has any discomfort radiating to his neck, jaw, arms, or back. If he does, ask him to describe it. Is it a dull, aching, pressurelike sensation? A sharp, stabbing, knifelike pain? Does he feel it on the surface or deep inside? Find out whether it’s constant or intermittent. If it’s intermittent, how long does it last? Ask if movement, exertion, breathing, position changes, or eating certain foods worsens or helps relieve the pain. Does anything in particular seem to bring it on?
Review the patient’s history for cardiac or pulmonary disease, chest trauma, intestinal disease, or sickle cell anemia. Find out which medications he’s taking, if any, and ask about recent dosage or schedule changes.
Take the patient’s vital signs, noting tachypnea, fever, tachycardia, oxygen saturation, paradoxical pulse, and hypertension or hypotension. Also, look for jugular vein distention and peripheral edema. Observe the patient’s breathing pattern, and inspect his chest for asymmetrical expansion. Auscultate his lungs for pleural friction rub, crackles, rhonchi, wheezing, and diminished or absent breath sounds. Next, auscultate for murmurs, clicks, gallops, and pericardial friction rub. Palpate for lifts, heaves, thrills, gallops, tactile fremitus, and abdominal masses or tenderness. (See Chest pain: Causes and associated findings, pages 164 to 167.)
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Signs & Symptoms (Fifth Edition), 2006
Pulse, absent or weak:
History and physical examination
(Professional Guide to Signs & Symptoms (Fifth Edition))
If you detect an absent or weak pulse, quickly palpate the remaining arterial pulses to distinguish between localized or generalized loss or weakness. Then quickly check other vital signs, evaluate cardiopulmonary status, and obtain a brief history. Based on your findings, proceed with emergency interventions. (See Managing an absent or weak pulse, pages 638 and 639.)
» READ BOOK EXCERPT ONLINE »
Source: Professional Guide to Signs & Symptoms (Fifth Edition), 2006
Chest Pain, Atypical:
History
(The 10-Minute Diagnosis Manual: Symptoms and Signs in the Time-Limited Encounter)
A. Characteristics of the chest pain. Important questions to ask: What is the quality of pain? Where is it located? What is its duration and intensity? What symptoms accompany the pain? Does anything trigger the chest pain or make the pain better or worse? Is there any relationship between exertion and the pain?
B. Determining the likelihood of ischemic heart disease. Four major features in the initial history and physical examination can be used to determine the likelihood of IHD. They are in order of importance:
1. Angina description (definite angina, probable angina, probably not angina, and not angina).
2. Prior myocardial infarction [by history, or electrocardiographic (EKG) findings].
3. Age (risk of IHD increases with age).
4. Number of risk factors (e.g., diabetes, smoking, hypercholesterolemia, and hypertension).
C. Features suggesting nonanginal pain. Features suggesting nonanginal pain include pleuritic pain (sharp or knifelike pain brought on by respiratory movements or cough), pain localized with one finger, pain reproduced by movement or palpation of the chest wall or arms, constant pain lasting for days, and very brief episodes of pain lasting a few seconds (1).
D. Other key considerations. Key considerations in the history include the following:
1. All presentations of chest pain should be taken seriously until proven to be benign.
2. The description of pain can be greatly influenced by socioeconomic status, education, culture, and personality.
3. A review of cardiac risk factors is appropriate for all patients who present with chest pain.
4. Red flags suggesting a noncardiac, life-threatening condition include tachypnea, dyspnea, and hypoxemia.
5. Sharp, stabbing, or pleuritic qualities do not completely exclude an ischemic cause (Chapter 8.5). In the Multicenter Chest Pain Study, IHD was diagnosed in 22% of patients coming to the emergency room with a sharp quality pain (2).
Physical examination
No reliable physical signs can be used to determine whether a patient with atypical chest pain has ischemic heart disease. The main purpose of the examination is to assess the patient for evidence of complications from atherosclerotic disease (e.g., peripheral vascular disease, cerebrovascular disease, and congestive heart failure). Pay attention to findings on the vascular examination (e.g., peripheral artery bruits, retinal arteriolar changes, the presence of a cardiac gallop) and for signs of the consequences of diminished myocardial contractility (e.g., lower extremity edema or pulmonary crackles) (Chapter 7.5).
» READ BOOK EXCERPT ONLINE »
Source: The 10-Minute Diagnosis Manual: Symptoms and Signs in the Time-Limited Encounter, 2000
Chest Pain, Substernal:
History
(The 10-Minute Diagnosis Manual: Symptoms and Signs in the Time-Limited Encounter)
A. Characteristics of pain in stable angina
1. Quality. The pain of angina pectoris is often not described as a pain at all. Instead, it is frequently referred to as a squeezing, heaviness, or pressure sensation lasting 5 to 10 minutes. Diaphoresis, dyspnea, nausea, and vomiting often accompany the discomfort. Pain that is sharp, stabbing (especially if exacerbated by deep inspiration), pain reproducible with chest wall palpation, and pain lasting seconds or days to weeks is less likely to be from CAD.
2. Location. Generally, angina is poorly localized in the retrosternal area, anterior chest, or epigastrium and typically radiates to the left arm, neck, or jaw.
3. Precipitating and alleviating factors. Angina is often precipitated by conditions that increase myocardial oxygen demand, most commonly physical exertion, emotional stress, or cold weather. It is relieved promptly with rest or sublingual nitroglycerin.
B. Characteristics of pain in unstable angina. According to the clinical practice guidelines recently developed by the Agency for Health Care Policy (1), unstable angina is defined as:
1. Angina at rest lasting greater than 20 minutes.
2. New onset angina (< 2 months) precipitated by walking one to two blocks or by climbing one flight of stairs at a normal pace.
3. Angina that is more frequent, longer in duration, or occurring at a lower threshold.
C. Risk factors. The Framingham Heart Study along with numerous other large epidemiologic studies has established the following risk factors for CAD (2):
1. Sex and age: men aged 45 years or older; women aged 55 years or older; women with premature menopause without hormone replacement.
2. Family history: MI or sudden death occurring in a first-degree male relative aged 55 years or younger or in a first-degree female relative aged 65 years or younger.
3. Smoking: in men who smoke one pack per day, a three- to fivefold risk for CAD compared with nonsmokers. Those who quit smoking can reach the same risk level of nonsmokers within 2 years of stopping.
4. Hypertension: blood pressure greater or equal to 140/90 (Chapter 7.8).
5. Cholesterol: total cholesterol greater than 200; low-density lipoprotein (LDL) greater than 130; high-density lipoprotein (HDL) less than 35. An HDL level above 60 is protective.
6. Diabetes mellitus: a twofold increase in CAD, compared with nondiabetics (Chapter 14.1).
Physical examination
A. Focused physical examination. This should include vital signs (notably blood pressure). During a symptomatic episode, the finding of a mitral regurgitation murmur, S3 or S4 gallop, bruits or precordial lift all suggest a high likelihood of CAD. Findings of xanthelasma, tendinous xanthomata, tobacco-stained teeth and fingernails, and decreased or asymmetrical peripheral pulses indicate the likely presence of cardiac risk factors.
>
» READ BOOK EXCERPT ONLINE »
Source: The 10-Minute Diagnosis Manual: Symptoms and Signs in the Time-Limited Encounter, 2000
Acute Nonpleuritic Chest Pain:
Differential Overview
(Field Guide to Bedside Diagnosis)
❑ Chest wall pain
❑ Angina
❑ Unstable angina
❑ Myocardial infarction
❑ Gastroesophageal reflux
❑ Herpes zoster
❑ Thoracic root compression
❑ Panic disorder
❑ Aortic stenosis
❑ Aortic dissection
❑ Mediastinal mass
❑ Biliary disease
Diagnostic Approach
It is essential to maintain a high index of suspicion (low threshold for investigation) for critical problems; however, most chest pain has a benign cause. The patient with myocardial ischemia often is reluctant to label the symptom as “pain.” Instead descriptors are used such as squeezing, pressure, tightness, fullness, a heavy weight on the chest, burning (attributed to indigestion),
or a toothache (when jaw radiation is present). A closed fist held to the sternum is commonly employed to explain the symptoms. Pleuritic chest pain, intensified by a deep breath, usually has a pulmonary or chest wall origin. Recurrent episodic pain or persistent pain lasting days is unlikely to represent a critical problem. Pain lasting a few seconds or pain that is sharp or stabbing in quality is almost never ischemic, especially if reproducible by palpation
or movement.
Syncope with chest pain should raise suspicion of aortic dissection,
ruptured aortic aneurysm, pulmonary embolism, or critical aortic stenosis. “Angor anomie,” a sense of impending doom, is found in serious conditions such as myocardial infarction, pulmonary embolism, aortic dissection, and to a lesser extent, panic disorder. Sternal pain may be caused by xiphoidalgia, myelomatosis, ankylosing spondylitis, osteomyelitis, or traumatic fracture.
» READ BOOK EXCERPT ONLINE »
Source: Field Guide to Bedside Diagnosis, 2007
Pleuritic Chest Pain:
Differential Overview
(Field Guide to Bedside Diagnosis)
❑ Costochondritis
❑ Pneumonia
❑ Rib fracture
❑ Pulmonary embolism
❑ Pleurisy
❑ Pneumothorax
❑ Pericarditis
❑ Lung cancer
❑ Pneumomediastinum
❑ Splenic infarction
Diagnostic Approach
Pleuritic chest pain, intensified by a deep breath, usually has a pulmonary or chest wall origin. Cardiac pain is almost never pleuritic (LR 0.2), sharp or stabbing (LR 0.3), positional (LR 0.3) or reproduced by palpation (LR 0.3).
» READ BOOK EXCERPT ONLINE »
Source: Field Guide to Bedside Diagnosis, 2007
Myocardial infarction:
Diagnosis
(Handbook of Diseases)
Persistent chest pain, ST-segment changes on the electrocardiogram (ECG), and elevated levels of total creatine kinase (CK) and the CK-MB isoenzyme over a 72-hour period usually confirm an MI. Cardiac troponins are useful in differentiating an MI from skeletal muscle injury, or when CK-MB measurements are low and a small MI has actually occurred. Auscultation may reveal diminished heart sounds, gallops and, in papillary dysfunction, the apical systolic murmur of mitral insufficiency over the mitral valve area.
When signs and symptoms are equivocal, assume that the patient has had an MI until tests rule it out. Diagnostic test results include the following:
❑ serial 12-lead ECG: ECG abnormalities may be absent or inconclusive during the first few hours following an MI. When present, characteristic abnormalities include serial ST-segment depression in subendocardial MI and ST-segment elevation in a transmural MI.
❑ coronary angiography: Visualization reveals which vessels have been affected and the extent of damage.
❑ serial serum enzyme levels: CK levels are elevated; specifically, CK-MB or troponin levels.
❑ myoglobin: Because myoglobin always rises within 3 to 6 hours after an MI, lack of an increase within 6 hours indicates that an MI hasn’t occurred.
❑ echocardiography: Echocardiography may show ventricular-wall motion abnormalities in patients with a transmural MI.
❑ nuclear ventriculography (multigated acquisition scan or radionuclide ventriculography) scanning: Nuclear scanning can identify acutely damaged muscle by picking up radioactive nucleotide, which appears as a “hot spot” on the film. It’s useful in localizing a recent MI.
Elevated homocysteine and C-reactive protein levels have been found incidentally in MI and may indicate a newer risk factor. The practical value of these tests remains unknown. Folic acid supplementation is used as treatment for elevated homocysteine levels.
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Coronary artery disease:
Diagnosis
(Handbook of Diseases)
The patient history — including the frequency and duration of angina and the presence of associated risk factors — is crucial in evaluating CAD. Additional diagnostic measures include the following:
❑ Electrocardiography (ECG) during angina may show ischemia or may be normal; it may also show arrhythmias, such as premature ventricular contractions. The ECG is apt to be normal when the patient is pain-free.
❑ Treadmill or bicycle exercise test may provoke chest pain and ECG signs of myocardial ischemia (ST-segment depression).
❑ Coronary angiography reveals narrowing or occlusion of the coronary artery, with possible collateral circulation.
❑ Myocardial perfusion imaging with thallium-201 or cardiolite during treadmill exercise detects ischemic areas of the myocardium, visualized as “cold spots.”
» READ BOOK EXCERPT ONLINE »
Source: Handbook of Diseases, 2003
Chest pain:
History
(Alarming Signs and Symptoms: Lippincott Manual of Nursing Practice Series)
Ask the patient to rate the pain using a standardized pain rating scale. Is the pain a dull, aching, pressurelike sensation, or sharp, stabbing, and knifelike? Is it constant or intermittent? If it’s intermittent, ask how long an episode lasts. Ask him about precipitating, aggravating, or alleviating factors. Review the patient’s history for cardiac or pulmonary disease, chest trauma, intestinal disease, or sickle cell anemia. Ask about medications he’s taking, if any, including recent dosage or schedule changes.
ALERT: Chest pain in perimenopausal women may be difficult to diagnose because it may present atypically. Fatigue, nausea, dyspnea, and shoulder or neck pain are symptoms more likely to signal an MI in women than in men.
Physical examination
Take the patient’s vital signs, noting tachypnea, fever, tachycardia, oxygen saturation, paradoxical pulse, and hypertension or hypotension. Check for jugular vein distention and peripheral edema. Observe the patient’s breathing pattern, and inspect his chest for asymmetrical expansion. Auscultate his lungs for pleural friction rub, crackles, rhonchi, wheezing, or diminished or absent breath sounds. Next, auscultate for murmurs, clicks, gallops, or pericardial friction rub. Palpate for lifts, heaves, thrills, gallops, tactile fremitus, and abdominal masses or tenderness.
» READ BOOK EXCERPT ONLINE »
Source: Alarming Signs and Symptoms: Lippincott Manual of Nursing Practice Series, 2007
Chest pain:
History
(Signs & Symptoms: A 2-in-1 Reference for Nurses)
If the chest pain isn’t severe, proceed with the history. Ask if the patient feels diffuse pain or can point to the painful area. Sometimes a patient won’t perceive the sensation he’s feeling as pain, so ask whether he has any discomfort radiating to his neck, jaw, arms, or back. If he does, ask him to describe it. Is it a dull, aching, pressurelike sensation? A sharp, stabbing, knifelike pain? Does he feel it on the surface or deep inside? Find out whether it’s constant or intermittent. If it’s intermittent, how long does it last? Ask if movement, exertion, breathing, position changes, or eating certain foods worsens or helps relieve the pain. Does anything in particular seem to bring it on?
Review the patient’s history for cardiac or pulmonary disease, chest trauma, intestinal disease, or sickle cell anemia. Find out which medications he’s taking, if any, and ask about recent dosage or schedule changes.
» READ BOOK EXCERPT ONLINE »
Source: Signs & Symptoms: A 2-in-1 Reference for Nurses, 2007
Pulse, absent or weak:
History
(Signs & Symptoms: A 2-in-1 Reference for Nurses)
After you detect an absent or weak pulse, review the patient’s history of heart disease. Ask him what medications he’s taking and whether he has any other illnesses. Also, question him about associated signs and symptoms, such as chest pain or dyspnea.
» READ BOOK EXCERPT ONLINE »
Source: Signs & Symptoms: A 2-in-1 Reference for Nurses, 2007
Chest Pain:
Clinical Features and Diagnosis
(The Diagnostic Approach to Symptoms and Signs in Pediatrics)
Musculoskeletal Disorders
Muscle
Trauma
Normal activitycan strain chest wall musculature.Participation in athletics or overexertionalso may injure specific muscle groups of chest wall.Blunt trauma from accidents, athleticinjuries, or physical abuse can cause chest wall contusions. Stitch
Definedas sharp pain occurring in upper quadrants of abdomen under costalmargin during strenuous activity.Resolves when exercise is over.Stress on peritoneal ligaments is thoughtto be the cause. Precordial Catch
Also calledTexidor twinge and defined as benign self-limited disorder of unknown cause.Characterized by acute onset of sharppain, usually localized over cardiac apex and occurring at restor with mild activity.Usually lasts up to 1 min but may befollowed by dull ache.Deep inspiration may aggravate pain. Sickle Cell Pain Episodes
Chest pain can occur during vasoocclusiveepisode and usually involves muscle ache.
Bone/Cartilage
Trauma
Accidentalor nonaccidental trauma may produce rib fractures.Localized bone tenderness suggestsrib contusion or fracture.Chest radiography should be performed. Costochondritis
Common causeof chest pain in adolescence.Localized pain and tenderness occurover the affected costochondral junction. Left fourth and fifthjunctions are most commonly involved. Sickle Cell Disease (Thoracic Bone Infarction)
Chest painmay occur as result of thoracic bone infarction that may affectribs, sternum, or vertebrae.Nuclear scintigraphy can demonstrateinfarction. Slipping-Rib Syndrome
Pain isthought to arise from eighth, ninth, or tenth ribs overriding theone above.Diagnosis may be confirmed by graspingcostal margin and pulling anteriorly to reproduce pain (hookingmaneuver). Same maneuver may produce palpable click as cartilagesslip over one another. Tietze Syndrome
Syndromeof unknown cause characterized by swelling at right sternoclavicularor second sternochondral junction.Pain may last for weeks with frequentremissions and exacerbations. Osteomyelitis
Localizedpain and tenderness along with fever characterize osteomyelitisof sternum or rib.S. aureus is most common pathogen.Nonspecific lab findings are leukocytosisand increased erythrocyte sedimentation rate.Although chest radiograph may be normalearly in illness, nuclear scintigraphy reveals localized uptakeof radionuclide.In 10–14 days, chest radiographyshows periosteal bone formation and destructive lesions.Bone aspiration or biopsy is usuallydiagnostic. Neoplasm
Bone tumorsof chest wall are rare; however, neoplastic disease may cause localized ribor sternal pain.Acute lymphoblastic leukemia may involvesternum or ribs. Chest pain is not infrequent during course of thisillness but is rarely presenting symptom. Metastatic neuroblastomaalso may cause bone pain.See Chap.1, Abdominal Masses and Chap. 38, Lymphadenopathy. Trachea and Proximal Bronchi Disorders
Tracheobronchialpain usually occurs in neck or chest where inflammation is.See Chap.10, Cough. Parietal Pleura Disorders
Intercostal nerves conduct pain impulsesfrom parietal pleura to spinal cord.
Pneumonia
Most commoncause of pleuritic chest pain is pneumonia.Pain is localized, sharp, stabbing,and knifelike and usually occurs with inspiration. Shallow breathsminimize pain, whereas laughing and coughing aggravate it. Pleuralfriction rub indicates pleural involvement.Chest radiograph shows infiltrate andsometimes pleural effusion.See Chap.10, Cough). Pleurodynia
Self-limitedillness usually caused by enteroviral infection.Fever and paroxysms of sharp chestpain without evidence of pneumonia are usual presenting clinicalfeatures. Pain usually subsides within 1 wk.Positive pharyngeal viral culture or4-fold increase in antibody titer is diagnostic. Empyema
Definedas presence of pus in pleural space and usually occurs from extensionof bacterial pneumonia.Most common pathogen in pediatric populationis S. pneumoniae.High spiking fever, respiratory distress,and occasional chest pain characterize empyema. Usually no breathsounds are heard over affected area.Chest radiograph shows effusion, butthoracentesis must be performed for specific diagnosis. Analysisof fluid should include white cell and differential counts; Gramand acid-fast stains; protein; glucose; and aerobic, anaerobic,fungal, and acid-fast cultures. Pneumothorax
Definedas accumulation of air in pleural space.Common causes include penetrating woundsof chest, rib fracture, positive pressure ventilation, pneumonia,cystic fibrosis, and idiopathic.Although chest pain is acute, unilateral,and severe, degree of respiratory distress depends on how largepneumothorax is. Large pneumothorax results in hyperresonance topercussion on involved side.Chest radiography is diagnostic. Hemothorax
Definedas collection of blood in pleural space.Most common cause is trauma, surgicalor nonsurgical.Degree of respiratory distress dependson size of hemothorax. Decreased breath sounds and dullness to percussionover affected side of chest usually occur.Chest radiograph that shows collapseof lung surrounded by opacification suggests presence of hemothorax.Diagnosis is confirmed by thoracentesis. Pneumomediastinum
Definedas presence of air or gas in mediastinum that can cause acute, severechest pain that may be referred to back, shoulders, and neck.Palpation of subcutaneous air in softtissues of chest wall or neck signifies presence of mediastinalemphysema.Common causes include pneumothoraxwith dissection of air into mediastinum, asthma, cystic fibrosis,and chest trauma with disruption of tracheobronchial tree.Chest radiography is diagnostic. Postpericardiotomy Syndrome
Thoughtto be immunologic reaction associated with introduction or reactivationof virus at time of intrapericardial surgery.Usually occurs 1–3 wks aftersurgery in children >2 yrs.Clinical manifestations include fever,chest pain, pericardial and pleural effusions, and sometimes pulmonaryparenchymal disease. Sharp or dull pain is precordial in locationand worsens with inspiration and lying down. Pleural and pericardialfriction rubs may be heard, unless there are significant effusions.Pericardial effusions can be life threateningif they are large.Serum may be positive for antiheartantibody.Average duration of illness is 2–3wks, but recurrences sometimes occur months or years later. Pulmonary Embolism
Definedas thrombus or any foreign material (e.g., air or fat) in pulmonaryarteries that causes obstruction of pulmonary blood flow.Risk factors in pediatric populationinclude deep venous thrombosis, central venous catheter, prolongedimmobilization, ventriculoatrial shunt, right-sided endocarditis,intravenous drug use, septicemia, and severe dehydration.Chest pain associated with pulmonaryembolism is acute and can be pleuritic or nonpleuritic. Usuallyassociated with dyspnea. Other findings include sweating, nausea,vomiting, palpitations, syncope, and anxiety.Physical exam commonly reveals tachycardia,dyspnea or tachypnea, and fever. Other findings include crackles,wheezes, pleural friction rub, prominent RV impulse, accentuatedpulmonary closure sound, S4 gallop, systolic ejection murmur alongsternal border, hepatomegaly, and edema.Chest radiograph is normal or showsconsolidation, atelectasis, or pleural effusion.ECG findings include ST-segment orT-wave changes in right precordial leads and RV hypertrophy.Pulmonary isotope lung perfusion scanthat shows bilateral segmental defects strongly suggests presenceof pulmonary emboli, whereas normal scan effectively excludes pulmonaryemboli. If scan is abnormal, ventilation (xenon) scan should beperformed. With pulmonary emboli, perfusion scan should be abnormaland ventilation scan normal.MRI also may be useful in diagnosisof pulmonary embolism. If diagnosis is still uncertain, pulmonaryangiogram is best way to establish presence of pulmonary embolism. Neoplasm
Primarychest neoplasms are rare in children.Chest pain is usually secondary topleural metastases, which can occur with Wilms tumor, osteogenicsarcoma, neuroblastoma, or rhabdomyosarcoma.Chest radiography may show mass ormetastases.Chest CT locates and defines extentof mass or metastases.Histologic diagnosis is definitive. Cardiac Disorders
Cardiacdisorders that cause chest pain usually produce myocardial ischemiaor inflammation of parietal pericardium.Middle and inferior cardiac nervesconduct myocardial pain impulses to upper thoracic rami, sympatheticchain, and spinal cord. Pain impulses from upper parietal pericardiumtravel along intercostal nerves, and those from lower pericardiumtravel along phrenic nerves and then to spinal cord. Myocardial Ischemia Including Infarction
Myocardialischemia causes angina, which has been described as gripping, crushing, sharp,knifelike pain in retrosternal or left chest areas that usuallyfollows exercise or exertion and is relieved by rest. Pain may betransmitted to neck, shoulder, arm, or back. In some cases, myocardialinfarction may occur. Tachypnea, tachycardia, gallop rhythm, sweating,nausea, and vomiting are common findings.Causes of myocardial ischemia includesevere aortic stenosis, coronary artery anomalies (including anomalouscoronary artery from pulmonary artery), myocarditis, cardiomyopathy,Kawasaki disease, familial dysproteinemias, and cocaine use.ECG changes may indicate myocardialischemia or injury. ECG leads overlying subepicardial or transmuralischemic area show inverted T waves. Symmetric tall peaked T wavesare seen in leads overlying subendocardial ischemic area. Leadsoverlying subendocardial injury show ST depression with concaveor flat contour, whereas those overlying subepicardial injury showST elevation with upwardly convex or concave contour. Within hoursto days of myocardial infarction, Q waves and inverted T waves appearin leads overlying infarction.2-D echocardiogram may show local orgeneralized myocardial dysfunction.Elevation of creatine kinase MB fractionor troponin T is usually found with destruction of myocardial tissue.Nuclear scintigraphy of myocardiummay show decreased local uptake.Cardiac catheterization and angiographyare necessary in some cases for definitive diagnosis. Pericarditis
Most commoncauses in children are viral infection, acute rheumatic fever, andpostpericardiotomy syndrome. Less common are purulent pericarditis,uremia, systemic lupus erythematosus, juvenile rheumatoid arthritis,and radiation therapy.Triad of fever, chest pain, and pericardialfriction rub signify pericarditis.Chest pain is usually acute and substernalwith radiation to neck, shoulders, and arms. Sitting up and leaningforward eases pain, whereas deep breathing, coughing, and lyingdown aggravate it.ECG shows elevated ST segments in nearlyall leads that progress to T-wave flattening and inversion in someof the leads, which may persist for months after acute lesion hasresolved.2-D echocardiography commonly revealspericardial effusion. Mitral Valve Prolapse
Associationof chest pain and mitral valve prolapse has been seriously questioned, bothin adults and children. Mechanism of pain, which is ill definedand usually nonexertional, is uncertain. Other causes of chest painshould be considered in children with chest pain and mitral valveprolapse.Midsystolic click or late systolicmurmur (mitral incompetence) or both are heard at apex.M-mode or 2-D echocardiography confirmsdiagnosis of mitral valve prolapse. Arrhythmias
Supraventriculartachycardia may cause some chest discomfort as well as cardiac failure.Sinus tachycardia or premature ventricularcontractions have been associated with palpitations.Important to differentiate by historywhether child is having chest pain or different sensation causedby palpitations.ECG rhythm strip confirms diagnosisof arrhythmia. If cardiac rhythm is normal, Holter monitoring maybe useful.With intermittent chest pain and suspectedarrhythmia, event recorder is another useful diagnostic technique. Diaphragm Disorders
Intercostalnerves conduct impulses from peripheral diaphragm to spinal cord. Phrenicnerves (C3–C5) conduct pain impulses from central diaphragmto spinal cord.With diaphragmatic irritation, painmay radiate to lower chest or shoulder, depending on extent of involvement.Possible causes include subphrenicor hepatic abscess and perihepatitis (Fitz-Hugh-Curtis syndrome). Gastrointestinal Disorders
Esophagus
Gastroesophageal Reflux
Common causeof transient chest pain is reflux of gastric contents from stomachinto esophagus. Severe reflux may lead to esophagitis with persistentchest pain.Monitoring for 24 hrs with esophagealpH probe can determine presence and severity of reflux. Endoscopywith biopsy can diagnose esophagitis.See Chap.55, Regurgitation and Vomiting). Caustic Ingestion
Ingestionof caustic substances may cause sharp or burning pain in midsternaland lower chest.Esophagoscopy is diagnostic. Foreign Body
Foreignbody lodged in esophagus commonly causes choking, drooling, anddifficulty swallowing. Associated dull ache also may occur.Chronically impacted foreign body maycause esophagitis.If foreign body is radiopaque, maybe seen on chest radiograph. Otherwise, filling defect may be seenon esophagram.Esophagoscopy is diagnostic for chronicallyimpacted foreign body. Hiatal Hernia
Definedas sliding hernia with gastroesophageal junction lying above diaphragm.Symptoms of hiatal hernia are thoseof gastroesophageal reflux with epigastric and lower chest paincommonly occurring after meals.Sometimes large hiatal hernia may beseen on plain radiograph with retrocardiac mass extending to rightlateral chest wall.Upper GI series is diagnostic. Spasm
May causechoking episodes during feeding, difficulty in swallowing, and substernal chestache.May be related to stress, rapid eating,and drinking cold liquids.Upper GI series with video playbackcapability is diagnostic. Tear
Persistentsevere vomiting may produce acute esophageal tear, which causeschest pain along with hematemesis.Esophagoscopy is diagnostic. Referred Pain
Gastritis,peptic ulcer disease, cholecystitis, and pancreatitis may causereferred chest pain.See Chap.2, Abdominal Pain. Neurologic Disorders
Intercostal Nerve
Trauma
Injury to intercostal nerve may produce painin dermatome supplied by nerve.
Herpes Zoster Neuritis
Herpes zostercan cause painful, vesicular lesions along ≥1 dermatomes correspondingto intercostal nerves.Positive viral culture of lesion isconfirmatory. Dorsal Root
Trauma (fractures)or spinal disease (tumor, osteomyelitis, epidural abscess) can involvecervical or upper thoracic dorsal roots and cause chest pain.Pain often occurs with body motionor after coughing, sneezing, or laughing. Hypesthesia to pin prickor light touch may be found in affected dermatomes.Useful diagnostic tests include cervicaland thoracic spine radiographs, nuclear scintigraphy, CT, and MRI. Psychologic Disorders
Anxietywith or without hyperventilation, depression, school phobia, hypochondriasis,and conversion reactions are common causes of chest pain, especiallyin adolescence.Pain has no particular characteristicsand usually diminishes once patients can talk about problem andreceive reassurance that they are not seriously ill.Hyperventilation attacks are most commonin girls and may produce air hunger, dizziness, syncope, palpitations,and paresthesias.Generally, specific stressful situationthat is related to onset of chest pain can be identified.Family history of chest pain and occurrenceof recurrent somatic complaints (e.g., headache and abdominal pain)are also common.To make diagnosis of psychogenic chestpain, positive clinical psychologic evidence must exist.Onset of pain in association with stressfulemotional situation suggests that pain is manifestation of psychologicproblem.In general, psychologic symptoms donot occur in isolation but are accompanied by other signs of unhappinessand anxiety.Psychosocial history is most usefulclinical tool in making diagnosis of psychogenic chest pain. Idiopathic Chest Pain
Most commoncause of chest pain in childhood and adolescence is idiopathic.Such pain is nonspecific but may be recurrent or chronic.This is diagnosis of exclusion. Diagnostic Approach
Most commoncauses of chest pain in pediatric population are idiopathic, musculoskeletal,and psychologic.If complaint has been present for >6mos, organic cause is less likely.History and physical exam are diagnosticin many cases.Chest radiography should be performedwith localized rib or bone pain, any respiratory distress, or suspectedpulmonary disorder.With suspected heart disease, ECG shouldbe performed.2-D echocardiography may be necessary,depending on suspected diagnosis.Psychologic causes must be substantiatedby clinical psychologic evidence.Children in whom no definite causecan be found are more likely to complain recurrently, presumablyfor secondary gain.In adolescence, chest pain is frequentcomplaint, but it is usually benign. Knowledge about recent lifeevents and individual's beliefs about the symptom are importantin managing this problem.
» READ BOOK EXCERPT ONLINE »
Source: The Diagnostic Approach to Symptoms and Signs in Pediatrics, 2006
Chest pain:
History and physical examination
(Nursing: Interpreting Signs and Symptoms)
If the chest pain isn't severe, proceed with the history. Ask if the patient feels diffuse pain or can point to the painful area. Ask when the pain began and if the patient ever experienced this type of pain in the past. Sometimes a patient won't perceive the sensation he's feeling as pain, so ask whether he has any discomfort radiating to his neck, jaw, arms, or back. If he does, ask him to describe it. Is it a dull, aching, pressurelike sensation? A sharp, stabbing, knifelike pain? Does he feel it on the surface or deep inside? Ask him to rate the pain on a pain scale. Find out whether it's constant or intermittent. If it's intermittent, how long does it last? Ask if movement, exertion, breathing, position changes, or eating certain foods worsens or helps relieve the pain. Does anything in particular seem to bring it on?
Review the patient's history for cardiac or pulmonary disease, chest trauma, intestinal disease, or sickle cell anemia. Find out which medications he's taking, if any, and ask about recent dosage or schedule changes.
Take the patient's vital signs, noting tachypnea, fever, tachycardia, oxygen saturation, paradoxical pulse, and hypertension or hypotension. Place the patient on a cardiac monitor and evaluate his heart rhythm. Also, look for jugular vein distention and peripheral edema. Note the feel of his skin. Is it cool and clammy or warm and diaphoretic? Auscultate his chest for extra heart sounds. Observe the patient's breathing pattern, and inspect his chest for asymmetrical expansion. Auscultate his lungs for pleural friction rub, crackles, rhonchi, wheezing, or diminished or absent breath sounds. Next, auscultate for murmurs, clicks, gallops, or pericardial friction rub. Palpate for lifts, heaves, thrills, gallops, tactile fremitus, and abdominal masses or tenderness.
» READ BOOK EXCERPT ONLINE »
Source: Nursing: Interpreting Signs and Symptoms, 2007
Pulse, absent or weak:
History and physical examination
(Nursing: Interpreting Signs and Symptoms)
If you detect an absent or a weak pulse, quickly palpate the remaining arterial pulses to distinguish between localized or generalized loss or weakness. If localized, ask whether the patient has experienced or is presently experiencing pain in that area. Assess the limb for color and temperature. Then quickly check the patient's other vital signs, evaluate his cardiopulmonary status, and obtain a brief history. Based on your findings, proceed with emergency interventions. (See Managing an absent or a weak pulse, pages 498 and 499.)
» READ BOOK EXCERPT ONLINE »
Source: Nursing: Interpreting Signs and Symptoms, 2007
CHEST PAIN:
Approach to the Diagnosis
(Differential Diagnosis in Primary Care)
A possible myocardial infarction must be the first consideration in all
adults with acute chest pain, especially if there are significant
alterations of the vital signs. Consequently, serial ECGs, serial cardiac
enzymes, and
hospitalization will often be necessary. After this condition has been excluded, we can turn our
attention to the other possibilities. Arterial blood gases, chest x-ray, and
a lung scan may be ordered to exclude a pulmonary embolism. Pulmonary
angiography may be necessary in some cases. A chest x-ray may be ordered to
rule out pneumonia. Acute chest pain related to esophagitis is often
relieved by swallowing lidocaine viscus, an extremely useful tool in the
differential diagnosis. Relief of the pain with nitroglycerin under the
tongue or by spray will support the diagnosis of coronary insufficiency.
Tenderness of the costochondral junctions with relief on lidocaine injection
into the point of maximum tenderness suggests Tietze syndrome
(costochondritis). In cases of chronic chest pain, an exercise tolerance
test with thallium scan should be done to rule out coronary insufficiency or
myocardial infarction. It may be wise to do immediate coronary angiography
if the condition deteriorates so that balloon angiography, bypass surgery,
or reperfusion therapy may be initiated. Dissecting aneurysm is revealed by
CT scan or MRI of the chest.
» READ BOOK EXCERPT ONLINE »
Source: Differential Diagnosis in Primary Care, 2007
Thrombosis:
Thrombosis - DIAGNOSIS
(The 5-Minute Pediatric Consult)
- Phase 1: Perform complete history and examination; establish diagnosis using the appropriate radiographic study.
- Phase 2: Send initial laboratory studies (CBC, PT/aPTT, D-dimer, antithrombin) and begin anticoagulation therapy with unfractionated heparin or low-molecular-weight heparin.
- Patients with life- or limb-threatening thrombosis may require thrombolysis.
- Phase 3: Complete lab workup for hypercoagulable state; outpatient anticoagulation; follow thrombosis radiologically
» READ BOOK EXCERPT ONLINE »
Source: The 5-Minute Pediatric Consult, 2008
A heart attack is caused when a blockage forms in the heart's blood vessels. Knowing what causes a heart attack and how to prevent it could save...
After you've already suffered from a heart attack, you are at high risk for a second. Tune in to learn how to help prevent a second heart attack.
The statistics for heart disease in women are staggering. Yet many don't know they are at risk, nor do they know the signs and symptoms of...
Nearly 60 million Americans are diagnosed with heart disease every year and it's the leading cause of death in both men and women. But...
See full list of 38 related videos
» Next page: Signs of Heart attack
Rate This Website
What do you think about the features of this website?
Take our user survey and have your say:
Website User Survey
Medical Tools & Articles:
Next articles:
Tools & Services:
Medical Articles:
Forums & Message Boards
- Ask or answer a question at the Boards:
